[1] Bagheri N,Azadegan-Dehkordi F,Rahimian G,et al.Altered Th17 cytokine expression in helicobacter pylori patients with TLR4 (D299G) Polymorphism[J].Immunol Invest,2016,45(2):161-171.
[2] Miftahussurur M,Yamaoka Y.Helicobacter pylori virulence genes and host genetic polymorphisms as risk factors for peptic ulcer disease[J].Expert Rev Gastroent,2015,9(12):1535-1547.
[3] Bagheri N,Azadegan-Dehkordi F,Shirzad M,et al.Mucosal interleukin-21 mRNA expression level is high in patients with Helicobacter pylori and is associated with the severity of gastritis[J].Cent Eur J Immunol,2015,40:61-67.
[4] Salimzadeh L,Bagheri N,Zamanzad B,et al.Frequency of virulence factors in Helicobacter pylori-infected patients with gastritis[J].Microb Pathog,2015,80:67-72.
[5] Rahimian G,Sanei MH,Shirzad H,et al.Virulence factors of Helicobacter pylori vacA increase markedly gastric mucosal TGF-β1 mRNA expression in gastritis patients[J].Microb Pathog,2014,67:1-7.
[6] Thi Huyen Trang T,Thanh Binh T,Yamaoka Y.Relationship between vacA types and development of gastroduodenal diseases[J].Toxins,2016,8(6):182.
[7] Müller A,Oertli M,Arnold IC.H.pylori exploits and manipulates innate and adaptive immune cell signaling pathways to establish persistent infection[J].Cell Commun Signal,2011,9(1):1.
[8] Raju D,Hussey S,Ang M,et al.Vacuolating cytotoxin and variants in Atg16L1 that disrupt autophagy promote Helicobacter pylori infection in humans[J].Gastroenterology,2012,142(5):1160-1171.
[9] Hashempour T,Bamdad T,Bergamini A,et al.F protein increases CD4+ CD25+ T cell population in patients with chronic hepatitis C[J].Pathogens and Disease,2015,73(4):ftv022.
[10] Shevach EM.Mechanisms of foxp3+ T regulatory cell-mediated suppression[J].Immunity,2009,30(5):636-645.
[11] Miyara M,Yoshioka Y,Kitoh A,et al.Functional delineation and differentiation dynamics of human CD4+ T cells expressing the FoxP3 transcription factor[J].Immunity,2009,30(6):899-911. [12] Noack M,Miossec P.Th17 and regulatory T cell balance in autoimmune and inflammatory diseases[J].Autoimmu Rev,2014,13(6):668-677.
[13] Enk AH.Dendritic cells in tolerance induction[J].Immunol Lett,2005,99(1):8-11.
[14] Collison LW,Workman CJ,Kuo TT,et al.The inhibitory cytokine IL-35 contributes to regulatory T-cell function[J].Nature,2007,450(7169):566-569.
[15] Larussa T,Leone I,Suraci E,et al.Helicobacter pylori and T helper cells:Mechanisms of immune escape and tolerance[J].J Immunol Res,2015,2015.
[16] Kao JY,Zhang M,Miller MJ,et al.Helicobacter pylori immune escape is mediated by dendritic cell–induced Treg skewing and Th17 suppression in mice[J].Gastroenterology,2010,138(3):1046-1054.
[17] Serrano C,Wright SW,Bimczok D,et al.Downregulated Th17 responses are associated with reduced gastritis in Helicobacter pylori-infected children[J].Mucosal Immunol,2013,6(5):950-959.
[18] Robinson K,Kenefeck R,Pidgeon EL,et al.Helicobacter pylori- induced peptic ulcer disease is associated with inadequate regulatory T cell responses[J].Gut,2008,57(10):1375-1385.
[19] Kindlund B,Sjöling Å,Hansson M,et al.FOXP3-expressing CD4+ T-cell numbers increase in areas of duodenal gastric metaplasia and are associated to CD4+ T-cell aggregates in the duodenum of helicobacter pylori-infected duodenal ulcer patients[J].Helicobacter,2009,14(3):192-201.
[20] Jang TJ.The number of Foxp3-positive regulatory T cells is increased in Helicobacter pylori gastritis and gastric cancer[J].Pathol Res Pract,2010,206(1):34-38.
[21] García M,Bellosillo B,Sanchez-Gonzalez B,et al.Study of regulatory T-cells in patients with gastric malt lymphoma:influence on treatment response and outcome[J].PLoS One,2012,7(12):e51681.
[22] Iwaya Y,Kobayashi M,Momose M,et al.High levels of FOXP3+ regulatory T cells in gastric MALT lymphoma predict responsiveness to Helicobacter pylori eradication[J].Helicobacter,2013,18(5):356-362.
[23] Amedei A,Codolo G,Del Prete G,et al.The effect of Helicobacter pylori on asthma and allergy[J].J Asthma Allergy,2010,3:139-147. |
