[1] Zhi-Gang X, Giuseppe P, Minghua L, et al. Acid-sensing ion channels (ASICs) as pharmacological targets for neurodegenerative diseases[J]. Curr Opin Pharmacol, 2008, 8(1): 25-32. [2] Geborek P, Saxne T, Pettersson H, et al. Synovial fiuid acidosis correlates with radiological joint destruction in rheumatoid arthritis knee joints[J].J Rheumatol, 1989, 16(4):468-472. [3] Kolker SJ, Walder RY, Usachev Y, et al. Acid-sensing ion channel 3 expressed in type B synoviocytes and chondrocytes modulates hyaluronan expression and release[J]. Ann Rheum Dis, 2009, 69(5): 903-909. [4] Schultz DR, Harrington WJ Jr. Apoptosis: programmed cell death at a molecular level[J]. Semin Arthritis Rheum, 2003,32(6): 345-369. [5] Kim HA, Song YW. Apoptotic chondrocyte death in rheumatoid arthritis[J]. Arthritis Rheum, 1999, 42(7): 1528-1537. [6] Kubasiak LA, Hemandez OM, Bishopric NH, et al. Hypoxia and acidosis activate cardiac rnyocyte death through the Bcl-2 family protein BNIP3[J]. Proc Natl Acad Sci USA, 2002, 99(20): 12825-12830. [7] 张腾跃,陈飞虎,王念,等. 酸敏感离子通道阻断剂阿米洛利对佐剂性关节炎大鼠关节软骨细胞凋亡抑制作用的初步研究[J]. 中国临床药理学与治疗学,2010, 15(1): 41-46. [8] Julkunen P, Kiviranta P, Wilson W, et al. Characterization of articular cartilage by combining microscopic analysis with a fibril-reinforced finite-element model[J]. J Biomech, 2007, 40(8):1862-1870. [9] Julkunen P, Korhonen RK, Nissi MJ, et al. Mechanical characterization of articular cartilage by combining magnetic resonance imaging and finite-element analysis: a potential functional imaging technique[J]. Phys Med Biol, 2008, 53(9): 2425-2438. [10] Wu MH, Urban JP, Zhan FC, et al. Effect of extracellular pH on matrix synthesis by chondrocytes in 3D agarose gel[J]. Biotechnol Prog, 2007, 23(2): 430-434. [11] 袁凤来,陈飞虎,黄学应,等. 酸敏感离子通道在大鼠佐剂性关节炎关节软骨中的表达[J]. 中华风湿病学杂志,2008,12(5):321-324. |