[1] 张赟.TLR2在骨髓间充质干细胞修复缺氧缺血性脑损伤中的作用及机制[D].重庆:重庆医科大学,2013. [2] Smith AL,Alexander M,Rosenkrantz TS,et al.Sex differences in behavioral outcome following neonatal hypoia ischemia:Insights from a clinical meta-analysis and a rodent model of induced hypoxic ischemic brain injury[J].Exp Neurol,2014,254:54-67. [3] 王钰,尹晓娟.锂对新生儿缺血缺氧性脑损伤神经保护作用的研究进展[J].中国儿童保健杂志,2011,19(11),1002-1004. [4] Camins A,Crespo-Biel N,Junyent F,et al.Calpains as a target for therapy of neurodegenerative diseases:putative role of Lithium[J].Current Drug Metabolism,2009,10(5),433-447. [5] Li Q,Li H,Roughton K,et al.Lithium reduces apoptosis and autophagy after neonatal hypoxia-ischemia[J].Cell Death Dis,2010,1(7):56-59. [6] Can A,Schulze TG,Gould TD.Molecular actions and clinical pharmacogenetics of lithium therapy[J].Pharmacol Biochem Behav,2014,123:3-16. [7] Shin WJ,Gwak M,Baek CH,et al.Neuroprotective effects of lithium treatment following hypoxic-ischemic brain injury in neonatal rats[J].Childs NervSyst,2012,28(2):191-198. [8] Rybakowski JK.Response to Lithium in Bipolar Disorder:clinical and genetic findings[J].ACS Chem Neurosci,2014,5(6):413-421. [9] Keshavarz M,Emamghoreishi M,Nekooeian AA,et al.Increased bcl-2 protein levels in rat primary astrocyte Culture Following Chronic Lithium Treatment[J].Iran J Med Sci,2013,38(3):255-262. [10] Cabrera O,Dougherty J,Singh S,et al.Lithium protects against glucocorticoid induced neural progenitor cell apoptosis in the developing cerebellum[J].Brain Res,2014,1545:54-63. [11] 邵肖梅,叶鸿瑁,丘小灿.实用新生儿学[M].4版.北京:人民卫生出版社,2011:700-701. [12] Tang XJ,Xing F.Calcium-permeable AMPA receptors in neonatal hypoxic-ischemic encephalopathy[J].Biomed Rep,2013,1(6):828-832. [13] Pasquali L,Busceti CL,Fulceri F,et al.Intracellular pathways underlying the effects of lithium[J].Behav Pharmacol,2010,21(5-6):473-492. [14] Tian N,Kanno T,Jin Y,et al.Lithium potentiates GSK-3βactivity by inhibiting phosphoinositide 3-kinase-mediated Akt phosphorylation[J].Biochem Biophys Res Commun,2014,450(1):746-749. [15] Motoi Y,Shimada K,Ishiguro K,et al.Lithium and autophagy[J].ACS Chem Neurosci,2014,5(6):434-442. [16] Leeds PR,Yu F,Wang Z,et al.A new avenue for Lithium:intervention in traumatic brain Injury[J].ACS Chem Neurosci,2014,5(6):422-433. [17] Dzietko M,Derugin N,Wendland MF,et al.Delayed VEGF treatment enhances angiogenesis and recovery after neonatal focal rodent stroke[J].Transl Stroke Res,2013,4(2):189-200. [18] Chan CB,Ye K.Phosphoinositide 3-kinase enhancer (PIKE) in the brain:is it simply a phosphoinositide 3-kinase/Akt enhancer?[J].Rev Neurosci,2012,23(2):153-161. [19] Chiu CT,Chuang DM.Molecular actions and therapeutic potential of lithium in preclinical and clinical studies of CNS disorders[J].Pharmacol Ther,2010,128(2):281-304. [20] Fan M,Song C,Wang T,et al.Protective effects of lithium chloride treatment on repeated cerebral ischemia-reperfusion injury in mice[J].Neurol Sci,2015,36(2):315-321. [21] Xie C,Zhou K,Wang X,et al.Therapeutic benefits of delayed lithium administration in the neonatal rat after cerebral hypoxia-ischemia[J].PLoS One,2014,9(9):e107192. [22] Li H,Li Q,Du X,et al.Lithium-mediated long-term neuroprotection in neonatal rat hypoxia-ischemia is associated with antiinflammatory effects and enhanced proliferation and survival of neural stem/progenitor cells[J].J Cereb Blood Flow Metab,2011,1(10):2106-2115. [23] Chicha L,Smith T,Guzman R.Stem cells for brain repair in neonatal hypoxia-ischemia[J].Childs Nerv Syst,2014,30(1):37-46. [24] 王钰,尹晓娟.锂对缺氧状态下新生鼠神经干细胞损伤的影响[J].中国儿童保健杂志,2013,21(10):1045-1048. |